External tibial torsion and lower back pain

How can external tibial torsion and lower back pain possibly be related? Let’s take a quick look at the anatomy and see how.

knees neutral, note external rotation of the right foot and decreased progression angle

knees neutral, note external rotation of the right foot and decreased progression angle

Remember the external tibial torsion is present if we drop a plumbline from the tibial tuberosity and it passes between the first and second metatarsals or more medially. This increases the progression angle of the foot. This occurs due to “over rotation" of the lower extremity during development, often exceeding the 1.5 degrees per year of external rotation per year up to age 15 or occurring for a longer period of time, up to skeletal maturity. It can be uni or bilateral.

note when the foot is neutral, the knee points inward

note when the foot is neutral, the knee points inward

Often, due to the increased progression angle, people will try to "straighten their feet" (ie, decrees their progression angle) to move forward in the sagittal plane. This places the knees to the inside of the sagittal plane which causes medial knee fall and sometimes increased mid and forefoot pronation. This results in increased medial spin of the thigh bilaterally which increases the lumbar lordosis. Combine this with a sway back or anterior pelvic tilt and you have increased pressure on the lumbar facet joints. The facets are designed to carry approximately 20% of the load put in these circumstances are often called upon to carry the much more. This often results in facet imbrication and lower back pain. You can strengthen the abdomen all you like but if you do not change the attitude of the foot, a will often develop lower back pain, especially when the abs fatigue. Now think about if the deformity is unilateral; this will often cause asymmetrical rotation of the pelvis in a clockwise or counter clockwise direction.

So, what can you do you?

Since external tibial torsion is a "hard deformity", we can influence how the bone grows before skeletal maturity but after that will not change significantly with stretching or exercise.

  • You can teach them to walk with an increase in progression angle (ie “duck footed”). This will often keep the knee in the sagittal plane and can be surprisingly well tolerated

  • You can use a foot leveling orthotic or arch support to bolster the arch and change the mechanics of the foot, causing external rotation of the tibia which will often result in a decrease in progression angle in compensation while still keeping the knee in the sagittal plane

  • You could place a full length varus wedge in the shoe which, by inverting the foot, externally rotates the tibia which the person will often compensates for by decreasing there progression angle to keep the knee and the sagittal plane



Dr Ivo Waerlop, one of The Gait Guys



#tibialtorsion #lowbackpain #LBP #progressionangle





Sometimes it’s OK for “toes in“ squats

We hear from folks and also read on a lot of blogs and articles about whether your toes should be in or out for squats or other types of activities. The real answer is “it depends”.

What it depends on is the patient’s specific anatomy. That means we need to pay attention to knees and hips and things like femoral and tibial torsion‘s. It’s paramount to keep the knees in the sagittal plane, no matter what the lower extremity orientation is.

When somebody has external tibial torsion (i.e. when you drop a plumbline from there to view tuberosity it passes medial to the line between the second and third or second metatarsal) then having your feet and externally rotated position places the knees in sagittal plane. Having the patient go “toes in” with this type of anatomy will cause both knees to for medially and create patellofemoral tracking issues.

Likewise, like the patient in the video, (Yes, I know I say “external tibial torsion“ at the beginning of the video but the patient has internal tibial torsion as you will see from the remainder of the video) when somebody has internal tibial torsion (I.e. when you drop a plumbline from the tibial tuberosity it passes lateral to the second metatarsal or a line between the second and third metatarsal) you would need to point the toes inward to keep the knees in the sagittal plane as demonstrated in the video. You can also see in the video when her feet are placed “toes out“ they fall outside sagittal plane laterally which creates patellofemoral tracking issues like it was in this particular patient.

So, knees in or knees out? It depends…

Dr. Ivo Waerlop, one of The Gait Guys

#internaltibialtorsion #externaltibialtorsion #kneepain #kneesin #kneesout #squats #thegaitguys

Podcast 150: Subtalar joint control? Plus Heel raise effects on low back pain

Links to find the podcast:
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Our website is all you need to remember. Everything you want, need and wish for is right there on the site.
Interested in our stuff ? Want to buy some of our lectures or our National Shoe Fit program? Click here (thegaitguys.com or thegaitguys.tumblr.com) and you will come to our websites. In the tabs, you will find tabs for STORE, SEMINARS, BOOK etc. We also lecture every 3rd Wednesday of the month on onlineCE.com. We have an extensive catalogued library of our courses there, you can take them any time for a nominal fee (~$20).

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http://directory.libsyn.com/episode/index/id/10909609


Show notes


The HyProCure proceedure

https://images.search.yahoo.com/yhs/search;_ylt=AwrEeBmEH0RdlDUAiAUPxQt.;_ylu=X3oDMTByMjB0aG5zBGNvbG8DYmYxBHBvcwMxBHZ0aWQDBHNlYwNzYw--?p=hyprocure+sinus+tarsi+implant&fr=yhs-sz-001&hspart=sz&hsimp=yhs-001

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4621198/
https://www.ncbi.nlm.nih.gov/pubmed/21106413
https://www.ncbi.nlm.nih.gov/pubmed/29786228

High-heeled walking decreases lumbar lordosis.EdenyBaaklini et al.
https://www.sciencedirect.com/science/article/pii/S096663621730108X

The effect of high-heeled shoes on lumbar lordosis: a narrative review and discussion of the disconnect between Internet content and peer-reviewed literature. Brent S. Russell
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3206568/

Prolong Wearing of High Heeled Shoes Can Cause Low Back PainFarjad Afzal1* and Sidra Manzoor
https://pdfs.semanticscholar.org/afb4/641b8ed6450fcbdfa8ff99029d935c2bdc88.pdf

Relation between Wearing High-Heeled Shoes and Gastrocnemius and Erector Spine Muscle Action and Lumbar Lordosis. Cezar Augusto Souza Casarin
https://www.medscitechnol.com/download/index/idArt/892352

A flatter foot approach?
https://twitter.com/IzzyMoorePhD/status/1157034538192855041

Thoughts: titrate into speed work just like doing the same for longer and longer runs
Creating a "speed base"
https://www.fastrunning.com/?p=26410&preview=true

"monster walks"
Hip-Muscle Activity in Men and Women During Resisted Side Stepping With Different Band Positions. Lewis CL, et al. J Athl Train. 2018.
https://www.ncbi.nlm.nih.gov/m/pubmed/30615490/

Physical findings differ between individuals with greater trochanteric pain syndrome and healthy controls: A systematic review with meta-analysis.
Plinsinga ML1, Ross MH1, Coombes BK2, Vicenzino B3.
Musculoskelet Sci Pract. 2019 Jul 25;43:83-90. doi: 10.1016/j.msksp.2019.07.009. [Epub ahead of print]
https://www.ncbi.nlm.nih.gov/pubmed/31369906

The Fudge Factor

image credit: https://commons.wikimedia.org/wiki/File:Pieces_of_fudge_cut_from_a_slab,_April_2008_cropped.jpg

image credit: https://commons.wikimedia.org/wiki/File:Pieces_of_fudge_cut_from_a_slab,_April_2008_cropped.jpg

We know from experience that it is often easier to accomplish a task faster, rather than slower (like an exercise or skiing) because of the cortex “interpolating” or making its “best guess” as to what (based on past experience) is going to happen and in what order. There is a certain amount of guess work (or what we call “the fudge factor”) involved.

Walking at a slower speed (or performing an exercise at a slower speed for that matter) has increased muscular demands, than doing it more quickly. Here is one study that exemplifies that.

“These findings may reflect a relatively higher than expected demand for peroneus longus and tibialis posterior to assist with medio-lateral foot stability at very slow speeds”

Here, they thought muscular demands would be proportional to speed, increasing with increasing demands. Like many things, what we think is going to happen and what actually happens can be 2 different things : )


Dr Ivo Waerlop, one of The Gait Guys


#fudgefactor #corticalinterpolation #muscledemands #gait #gaitguys


Gait Posture. 2014 Apr;39(4):1080-5. doi: 10.1016/j.gaitpost.2014.01.018. Epub 2014 Feb 6.

Electromyographic patterns of tibialis posterior and related muscles when walking at different speeds.

Murley GS1, Menz HB2, Landorf KB2.

Botox for plantar fasciitis? Sounds like a bad idea to us....

image source: https://commons.wikimedia.org/wiki/File:Plantar_aponeurosis_-_axial_view.png

image source: https://commons.wikimedia.org/wiki/File:Plantar_aponeurosis_-_axial_view.png

Botox..For plantar fasciitis? Really?

We found this article (1) in one of our favorite journals, Lower Extremity Review , and were a little surprised. Let us get this straight: you are going to take one of the the most poisonous biological neurotoxins known (1) and inject it into your calf and foot?

The article in LER is well written and the results (thankfully) were inconclusive regarding its usage. They do cite 3 studies (with two by the same lead author) where it has been effective (2-4). Yes, it is better than saline (5) (but not as good as extracorporeal shock wave therapy (6)), and better than placebo (7-10) but considerably more risky.

So the premise is “if the muscle is dysfunctional, then let’s just take it out of the equation”. But this really doesn’t fix the problem, it just covers up the symptom. And what about the other potential side effects since botulinum toxin acts not only at the neuromuscular junction, blocking the release of acetylcholine, but also at the autonomic ganglia, postganglionic parasympathetic nerve endings, as well as the post ganglionic sympathetics that use acetylcholine (capillaries of skin, piloerector muscles and sweat glands) (11)?.

In our experience, most cases of plantar fasciitis are secondary to lack of forefoot rocker, lack of ankle rocker, lack of hip extension or in some cases, direct trauma. Wouldn’t it make more sense to strengthen the anterior compartment to reciprocally inhibit the posterior compartment, increasing ankle dorsiflexion and hip extension? We find, oftentimes, treating only the area of chief complaint and not what is "driving the bus" can offer temporary, symptomatic relief but not long standing pathmechanics or pathoanatomy.

Just like the road to enlightenment, there are no shortcuts in treating plantar fasciitis and if you are not going to treat the cause, then be prepared to reap what you sow.

Dr Ivo Waerlop, one of The Gait Guys

#botox #plantarfascitis #badideas #gaitproblem #thegaitguys

1. https://lermagazine.com/article/botox-injection-not-just-for-celebrities-furrows-and-wrinkles

2. Elizondo-Rodriguez J, Araujo-Lopez Y, Moreno-Gonzalez JA, Cardenas-Estrada E,
Mendoza-Lemus O, Acosta-Olivo C. A comparison of botulinum toxin A and intralesional steroids for the treatment of plantar fasciitis: A randomized, double-blinded study. Foot Ankle Int.
2013;34(1):8-14.

3. Díaz-Llopis IV, Rodríquez-Ruíz CM, Mulet-Perry S, Mondéjar-Gómez FJ., Climent-Barberá JM., Cholbi-Llobel F. Randomized controlled study of the efficacy of the injection of botulinum toxin type A versus corticosteroids in chronic plantar fasciitis: results at one and six months. Clin Rehabil. 2012;26(7):594-606.

4. Díaz-Llopis IV, Gómez-Gallego D, Mondéjar-Gómez FJ, López-García A, Climent-Barberá JM, Rodríguez-Ruiz CM. (2013). Botulinum toxin type A in chronic plantar fasciitis: clinical effects one year after injection. Clin Rehabil. 2013;27(8):681-685.

5. Ahmad J, Ahmad SH, Jones K. Treatment of Plantar Fasciitis With Botulinum Toxin. Foot Ankle Int. 2017 Jan;38(1):1-7. doi: 10.1177/1071100716666364. Epub 2016 Oct 1.1.

6. Roca B, Mendoza MA, Roca M. Comparison of extracorporeal shock wave therapy with botulinum toxin type A in the treatment of plantar fasciitis. Disabil Rehabil. 2016 Oct;38(21):2114-21. doi: 10.3109/09638288.2015.1114036. Epub 2016 Mar 1

7. Babcock MS, Foster L, Pasquina P, Jabbari B. Treatment of pain attributed by plantar fasciitis with botulinum toxin A: a short-term randomized, placebo-controlled, double blinded study. Am J Phys Med Rehabil. 2005;84(9):649-654.

8. Samant PD, Kale SY, Ahmed S, Asif A, Fefar M, Singh SD. Randomized controlled study comparing clinical outcomes after injection botulinum toxin type A versus corticosteroids in chronic plantar fasciitis. Int J Res Orthop. 2018;4(4):672-675.

9. Huang YC, Wei SH, Wang HK, Lieu FK. Ultrasonographic guided botulinum toxin type A treatment for plantar fasciitis: an outcome-based investigation for treating pain and gait changes. J Rehabil Med. 2010;42(2):136-140.

10. Ahmad J, Ahmad SH, Jones K. Treatment of plantar fasciitis with botulinum toxin. Foot Ankle Int. 2017;38(1):1-7.

11. Nigam PK, Nigam A. Botulinum toxin. Indian J Dermatol. 2010;55(1):8–14. doi:10.4103/0019-5154.60343

Low back pain and asymmetry.

Screen Shot 2019-04-07 at 10.00.54 AM.png

Do oarsmen have asymmetries in the strength of their back and leg muscles?
IF these oarsmen were more symmetrical would they not be in pain?

From the study below:
"Patterns of asymmetry of muscle activity were observed between the left and right erector spinae muscles during extension, which was significantly related to rowing side (P < 0.01). These observations could be related to the high incidence of low back pain in oarsmen."

Here we have a supported study of asymmetry and injury/pain. This is what we have been saying (asymmetry matters) in the last few days with our posts on asymmetry. This study eludes to a finding that strength can test normal and symmetrical, but EMG activity can show patterns of asymmetry that can result in problems/pain.

Have you ever rowed? I mean truly rowed, in a shell, on the water, not on land or on a Concept 2 rower? It is just not the same, especially if you have an unilateral asymmetrical loading arc, like an oarsman pulling from port or starboard. I have rowed on the water just like this, briefly, one summer in a camp for young teens. I rowed on my home town course, on the World famous Royal Canadian Henley Regatta. I was the 2nd seat, starboard, in an 8 man shell. 8 oars in the water, 8+1 guys, one oar a piece, alternating port and starboard. I was behind the stroke. I hated it. Perhaps the hardest thing I had ever done sport wise to that point, largely because this dude setting the pace was jacked on caffeine, or something else, I think. No one works harder than rowers if you ask me, they are some of the fittest athletes in the world. Why? because it is a whole body effort.
Ok, enough of the fluff.

Now imagine rowing like this for many years in high school, college and/or competitively. Forcefully pulling on one oar, across an arc of pull out one side of the boat, thousands of times a day for many years. If that isn't something that will develop asymmetry I do not know what might. Oarsman are under near constant high end effort pushing and pulling loads (push with the legs, pull with the arms). There are few, if any, sports with such high end constant effort than rowing.

From the Parkin et al study:
"The aim of this study was to establish whether asymmetry of the strength of the leg and trunk musculature is more prominent in rowers than in controls. Nineteen oarsmen and 20 male controls matched for age, height and body mass performed a series of isokinetic and isometric strength tests on an isokinetic dynamometer. These strength tests focused on the trunk and leg muscles. Comparisons of strength were made between and within groups for right and left symmetry patterns, hamstring: quadriceps ratios, and trunk flexor and extensor ratios. The results revealed no left and right asymmetries in either the knee extensor or flexor strength parameters (including both isometric and isokinetic measures). Knee extensor strength was significantly greater in the rowing population, but knee flexor strength was similar between the two groups. No difference was seen between the groups for the hamstring: quadriceps strength ratio. In the rowing population, stroke side had no influence on leg strength. No differences were observed in the isometric strength of the trunk flexors and extensors between groups, although EMG activity was significantly higher in the rowing population. Patterns of asymmetry of muscle activity were observed between the left and right erector spinae muscles during extension, which was significantly related to rowing side (P < 0.01). These observations could be related to the high incidence of low back pain in oarsmen."- Parkin et al.

Extra sauce:
I "caught a crab" many times when a novice oarsman and was nearly vaulted out of the boat on one fatal event. A crab is the term rowers use when the oar blade gets “caught” in the water. It is caused by a momentary flaw in oar technique and the paddle end of the oar is pulled into the depths instead of skimming just below the surface. Catching a crab has happened to anyone who has ever rowed. A crab may be minor, allowing the rower to quickly recover, or it may be so forceful that the rower is ejected from the boat as the handle end catches the oarsman under the arms lifting them out of the boat.

J Sports Sci. 2001 Jul;19(7):521-6.
Do oarsmen have asymmetries in the strength of their back and leg muscles? Parkin S1, Nowicky AV, Rutherford OM, McGregor AH.